Re: Somalis Eating Pork
Posted: Sat Mar 29, 2008 5:58 pm
FAT AND CANCER
During the past five decades, the influence of dietary fat on the development of certain forms of cancer has led to the conclusion that there is a good positive correlation between the dietary pork fat consumption and cancer rates of the colon, breast, prostate. endometrium, pancreas, and of the biliary system.
These correlations have arisen from three kinds of evidence. First, descriptive epidemiologic studies which focus on the effect of fat, especially lard, on cancer incidence and mortality rates among different countries and among population groups who have different fat intake and different dietary habits such as the Seventh-Day Adventists. Second, evidence came from experimental studies. These experimental studies have been assisted by the discovery of several animal models in which a lesion mimicing human lesionscan be induced chemically. In these models, animals which have been fed a high lard diet developed more colon and breast tumors than animals fed low lard diets.
Third, strong evidence relating the etiology of the above-mentioned cancer to lard came from migrant studies. Within two to three generations, Japanese migrants to the U.S.A. experience an increase in cancer incidence rates. Breast cancer in Japanese-American women had risen to five times that of age-matched native Japanese women during the years 1969-1971 .The same is true for the Police immigrants to the United States These migrant studies exclude the possibility of genetic variations.
Current evidence indicates that the possible mechanisms by which dietary pork fat could playa promoting effect in human carcinogenesis could be through: 1) its effects on the production, activation, or inactivation of carcinogens by the intestinal flora, 2) its effect on the endogenous production, activation, or inactivation of carcinogens or 3) its effect on tissues to alter their susceptibility to carcinogenesis.
Correlation studies between different sources of fat and breast cancer concluded that the highest positive correlation was found for pork fat, followed by other animal fat intake and that a similar association could not be found for vegetable fat.
During the past five decades, the influence of dietary fat on the development of certain forms of cancer has led to the conclusion that there is a good positive correlation between the dietary pork fat consumption and cancer rates of the colon, breast, prostate. endometrium, pancreas, and of the biliary system.
These correlations have arisen from three kinds of evidence. First, descriptive epidemiologic studies which focus on the effect of fat, especially lard, on cancer incidence and mortality rates among different countries and among population groups who have different fat intake and different dietary habits such as the Seventh-Day Adventists. Second, evidence came from experimental studies. These experimental studies have been assisted by the discovery of several animal models in which a lesion mimicing human lesionscan be induced chemically. In these models, animals which have been fed a high lard diet developed more colon and breast tumors than animals fed low lard diets.
Third, strong evidence relating the etiology of the above-mentioned cancer to lard came from migrant studies. Within two to three generations, Japanese migrants to the U.S.A. experience an increase in cancer incidence rates. Breast cancer in Japanese-American women had risen to five times that of age-matched native Japanese women during the years 1969-1971 .The same is true for the Police immigrants to the United States These migrant studies exclude the possibility of genetic variations.
Current evidence indicates that the possible mechanisms by which dietary pork fat could playa promoting effect in human carcinogenesis could be through: 1) its effects on the production, activation, or inactivation of carcinogens by the intestinal flora, 2) its effect on the endogenous production, activation, or inactivation of carcinogens or 3) its effect on tissues to alter their susceptibility to carcinogenesis.
Correlation studies between different sources of fat and breast cancer concluded that the highest positive correlation was found for pork fat, followed by other animal fat intake and that a similar association could not be found for vegetable fat.